Glycolytic Phenotype and AMP Kinase Modify the Pathologic Response of Tumor Xenografts to VEGF Neutralization
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Nevena Djuranovic
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Giorgia Nardo, Elena Favaro, Matteo Curtarello, Lidia Moserle, Elisabetta Zulato, Luca Persano, Elisabetta Rossi, Giovanni Esposito, Marika Crescenzi, Oriol Casanovas, Ulrike Sattler, Wolfgang Mueller-Klieser, Barbara Biesalski, Oliver Thews, Rossella Canese, Egidio Iorio, Paola Zanovello, Alberto Amadori, and Stefano Indraccolo
Isolation and expansion of regionally defined human glioblastoma cells in vitro
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Nevena Djuranovic
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Pistollato F1, Persano L, Puppa AD, Rampazzo E, Basso G.
Hypoxia Selects Bortezomib-Resistant Stem Cells of Chronic Myeloid Leukemia
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Nevena Djuranovic
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Michele Tanturli, Serena Giuntoli, Valentina Barbetti, Elisabetta Rovida, Persio Dello Sbarba
Glucose availability in hypoxia regulates the selection of chronic myeloid leukemia progenitor subsets with different resistance to imatinib-mesylate
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Nevena Djuranovic
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Serena Giuntoli, Michele Tanturli, Federico Di Gesualdo, Valentina Barbetti, Elisabetta Rovida, and Persio Dello Sbarba
Interaction of hypoxia-inducible factor-1α and Notch signaling regulates medulloblastoma precursor proliferation and fate
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Nevena Djuranovic
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Pistollato F, Rampazzo E, Persano L, Abbadi S, Frasson C, Denaro L, D’Avella D, Panchision DM, Della Puppa A, Scienza R,Basso G.
Hypoxia and succinate antagonize 2-deoxyglucose effects on glioblastoma.
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Nevena Djuranovic
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Pistollato F, Abbadi S, Rampazzo E, Viola G, Della Puppa A, Cavallini L, Frasson C, Persano L, Panchision DM, Basso G.
Hypoxia and mitochondrial oxidative metabolism
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Nevena Djuranovic
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G Solaini, A Baracca, G Lenaz, G Sgarbi
Hypoxia suppresses BCR/Abl and selects imatinib-insensitive progenitors within clonal CML populations
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Nevena Djuranovic
Read more about Hypoxia suppresses BCR/Abl and selects imatinib-insensitive progenitors within clonal CML populations
Giuntoli S, Rovida E, Barbetti V, Cipolleschi MG, Olivotto M, Dello Sbarba P